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Baicalin has been reported to improve liver injury and fibrosis by suppressing transforming growth factor beta1 (TGFβ1), a major fibrogenic inflammatory cytokine, and activating peroxisome proliferator-activated receptor γ (PPARγ), a nuclear receptor with protective effect in fibrosis (Qiao et al., 2011)

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We are all well aware that high levels of homocysteine are associated with a wide range of diseases, as shown in Table 1